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Author: search.filters.author.Baloski MarjanSubject: search.filters.subject.pulmonary embolism

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    PLUCNA EMBOLIJA U BOLESNIKA S LEIDENOVOM TROMBOFILIJOM FAKTORA V I PSORIJAZOL: PRIKAZ SLUCAJ.
    (Association of pulmologists from Republika Srpska, 2023-05)
    Baloski Marjan
    ;
    ;
    Bushev Jane
    ;
    Brishkoska-Boshkovski Vesna
    ;
    Hasan Taner
    Cilj: Genetski faktori rizika pove!avaju rizik venske tromboembolije. Poremecaji u sintezi ili aktivnosti faktora koagulacije. Faktor V Leiden, protrombin (20210-A), antitrombinski deficit, deficit proteina C i proteina S i hiperhomocisteinemija najcesce su mutacije gena povezanih sa venskim tromboembolijom . Uvod : Psorijaza i prisutvo mutacije trombofilnih gena povecava rizik venske tromboembolije. Prethodna venska tromboembolija je jedan od najjacih faktora rizika, cak i kod pacijenata koji su aktivno leceni antikoagulansom. Psorijaza je kompleksna imuno posredovana bolest, povezana sa kardiovaskularnim rizikom, markerima hiperkoagulabilnosti i povisenim homocisteinom. Mnogo izvjestaja o opservacijama sugerira povecanu ucestalost venskih trombembolickih dogadaja kod pacijenata sa psorijazom. Nalazi: Prikazujemo bolesnika s nasljednom trombofilijom i kronicnom difuznom psorijazom kompliciranom plu!nom embolijom. Analiza DNK ukazuje na prisutnost homozigoze za mutaciju faktora V Leidena. Dermatoloska anamneza je pozitivna na psorijazu. Zaklju!ak: Prikaz ovog slucaja ukazuje na povezanost venske tromboembolije i psorijaze. Pacijenti sa naslednom trombofilijom, psorijazom I plucnim tromboembolizmom, imaju visoki rizik od razvoj venske tromboembolije.
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    DESCRIPTIVE ANALYSIS OF CLINICAL AND DEMOGRAPHIC DATA OF SELECTED GROUP OF PATIENTS WITH PULMONARY EMBOLISM
    (Macedonian Association of Anatomists and Morphologists, 2020)
    Baloski Marjan
    ;
    ;
    Hasan Taner
    ;
    Nedeska Minova Natasa
    ;
    Pulmonary embolism is a relatively common clinical entity accompanied with a high mortality and is a perplexing diagnostic and therapeutic problem. Current research indicates that pulmonary embolism has a multifactorial and complex pathogenesis. Genetic factors have been under extensive research during the past two decades. The aim of this study was to present a descriptive analysis of demographic and clinical data obtained thus far from 31 patients with documented pulmonary embolism. In our patient group, almost half of the patients were non-smokers and all denied alcohol consumption. More than 80% of the patients had no history of previous pulmonary embolism and no thrombophlebitis, but nearly two thirds of all patients had deep vein thrombosis. A history of acute myocardial infarction existed in about 6.5% of patients, as well as ischemic stroke. Arterial hypertension was present in about one-third of patients, dyslipidemia in 42%, and type 2 diabetes in approximately 13%. Only one patient had an anamnestic data for chronic renal disease, while none had a history of hepatic disease. The results of the analysis of demographic-clinical data of patients are concordant with the results of the previously published studies.