Ivanovski, Ognen
Preferred name
Ivanovski, Ognen
Official Name
Ivanovski, Ognen
Alternative Name
O ivanovski
Ognen, Ivanovski
Ivanovski o
Ivanovski ognen
Ivanovski, Ognen
Ognen Ivanovski
Ivanovski, O
Main Affiliation
Email
ognen.ivanovski@medf.ukim.edu.mk
Scopus Author ID
9246276600
Researcher ID
B-4025-2008
76 results
Now showing 1 - 10 of 76
- Some of the metrics are blocked by yourconsent settings
Item type:Publication, S128 Different approaches in buccal mucosa urethral augmentation-dorsal or ventral onlay, dorsal inlay or two stage urethroplasty?(Elsevier BV, 2013-10); ; ;Shabani, B. ;Trajkovski, D.Djordjevic, M. - Some of the metrics are blocked by yourconsent settings
Item type:Publication, The antioxidant N-acetylcysteine prevents accelerated atherosclerosis in uremic apolipoprotein E knockout mice(Elsevier BV, 2005-06); ;Szumilak, Dorota ;Nguyen-Khoa, Thao ;Ruellan, NadyaPhan, OlivierCardiovascular disease is the most frequent cause of mortality in chronic renal failure (CRF). Therefore, it is important to identify appropriate treatment measures. The antioxidant N-acetylcysteine (NAC) has been shown to reduce cardiovascular events in hemodialysis patients. Here we examine a possible direct effect of NAC supplementation on uremia-enhanced atherosclerosis in apolipoprotein E-deficient (apoE(-/-)) mice. - Some of the metrics are blocked by yourconsent settings
Item type:Publication, POS-01.85: Penile strangulation: presentation of two unusual cases(Elsevier BV, 2007-09); ; ;Kuzmanoski, M. ;Banev, S.Lekovski, L. - Some of the metrics are blocked by yourconsent settings
Item type:Publication, S178 EXTRACORPOREAL SHOCKWAVE LITHOTRIPSY IN THE TREATMENT OF RENAL AND URETERAL STONES: A MULTICENTER INTERNATIONAL EXPERIENCE WITH SIEMENS LITHOSKOP®(Elsevier BV, 2012-10) ;Penev, M. ;Stefanovsky, A. ;Batandjiovski, T. ;Stankov, O.Stojanoski, I. - Some of the metrics are blocked by yourconsent settings
Item type:Publication, Evaluation of the value of p53 protein expression in the extra-capsular extension of prostate cancer(2011); ;Georgiev, V; ;Penev, MThe objective of this study is to identify the nuclear expression of the p53 protein in prostate cancer and to determine its relationship with clinico-pathological variables. - Some of the metrics are blocked by yourconsent settings
Item type:Publication, Atherosclerosis and vascular calcification in uraemia - a new experimental model(Macedonian Academy of Sciences and Arts, 2007-12); ; ;Drueke, B TMassy, A ZCardiovascular disease (CVD) is the most frequent cause of morbidity and mortality in chronic renal failure (CRF) patients. Accelerated calcifying atherosclerosis, medial calcification, and valvular calcification are hallmarks of CVD in the dialysis population. The mechanisms by which uraemia promotes vascular calcification and the relationship between arterial wall calcification and atherosclerosis are poorly understood. We surgically induced CRF in apolipoprotein E knockout (apoE-/-) mice to study a possible acceleration of aortic atherosclerosis, the degree and type of vascular calcification as well as factors involved in the calcification process. Finally we investigated appropriate treatment measures. Atherosclerotic lesions in the thoracic aorta were significantly larger in uraemic apoE-/- mice than in non-uraemic controls. The relative proportion of the calcified area to the total surface area of both atherosclerotic lesions and lesion-free vascular tissue was increased in the aortic root of uraemic apoE-/- mice when compared with controls. The accelerated atherosclerosis was associated with an increase in aortic nitrotyrosine expression, indicating enhanced oxidative stress, and an increase in plaque collagen content, indicating changes in plaque composition. N-acetylcysteine (NAC) treatment slowed the rapid progression of atherosclerotic lesions and reversed the increase in plaque collagen content compared with placebo treatment. NAC-treatment also reduced nitrotyrosine expression in uremic apoE-/- mice whereas the degree of macrophage infiltration was unchanged. Sevelamer treatment delayed not only vascular calcification but also atherosclerotic lesion progression in uraemic apoE-/- mice. These treatment effects also were associated with diminished oxidative stress and were independent of cholesterol lowering. We anticipate that this experimental model will prove to be useful to test other treatment strategies aimed at decreasing the accelerated atherosclerosis and arterial calcification of the uraemic state. - Some of the metrics are blocked by yourconsent settings
Item type:Publication, A new era in the treatment of calcium oxalate stones?(Elsevier BV, 2013-06); Drüeke, Tilman BCalcium oxalate (CaOx) is the most prevalent type of kidney stone. The amount of oxalate excreted in the urine is a major risk factor for CaOx stone formation. The study by Siener et al. makes a substantial contribution to our understanding of how Oxalobacter formigenes affects oxalate metabolism and excretion in humans and hence influences the risk of developing CaOx kidney stones. - Some of the metrics are blocked by yourconsent settings
Item type:Publication, - Some of the metrics are blocked by yourconsent settings
Item type:Publication, S54 CHEMICAL COMPOSITION OF URINARY TRACT STONES IN REPUBLIC OF MACEDONIA(Elsevier BV, 2011-10) ;Davceva, O. ;Nikolov, G. - Some of the metrics are blocked by yourconsent settings
Item type:Publication, Calcification of the cavernosal bodies may be responsible for development of erectile dysfunction in uremic apolipoprotein E deficient (apoE-/-) mice(Elsevier BV, 2019-12-30); ; ;Davceva, OliveraErectile dysfunction's physiopathology in uremia is complex and multifactorial, involving a combination of classical risk factors and specific uremia-related risk factors such as increased oxidative stress, endothelial dysfunction and inflammation. The aim of the study is to investigate the effect of chronic kidney disease (CKD) on vascular calcification and endothelial function of cavernosal bodies in apolipoprotein E deficient (apoE-/-) mice, a well known model of erectile dysfunction.
