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Please use this identifier to cite or link to this item: http://hdl.handle.net/20.500.12188/32056
DC FieldValueLanguage
dc.contributor.authorSelimov, Pavelen_US
dc.contributor.authorKaralilova, Rositsaen_US
dc.contributor.authorDamjanovska Krstikj, LJubinkaen_US
dc.contributor.authorDelcheva, Ginkaen_US
dc.contributor.authorStankova, Teodoraen_US
dc.contributor.authorStefanova, Katyaen_US
dc.contributor.authorManeva, Anaen_US
dc.contributor.authorSelimov, Teodoren_US
dc.contributor.authorBatalov, Anastasen_US
dc.date.accessioned2024-12-16T13:37:17Z-
dc.date.available2024-12-16T13:37:17Z-
dc.date.issued2023-02-28-
dc.identifier.urihttp://hdl.handle.net/20.500.12188/32056-
dc.description.abstractRheumatoid arthritis (RA) is the most common inflammatory joint disease. Various proinflammatory cytokines are involved in the pathogenesis of this chronic disorder. It is characterized by the presence of autoantibodies, such as rheumatoid factor and antibodies against citrullinated peptides. The present study focuses on investigation of possible association between the proinflammatory cytokine interleukin 17 and anti-CCP, anti-MCV, and anti-CarP antibodies seropositivity in RA patients.en_US
dc.language.isoenen_US
dc.publisherPensoft Publishersen_US
dc.relation.ispartofFolia Medicaen_US
dc.subjectanti-CCPen_US
dc.subjectanti-MCVen_US
dc.subjectanti-CarPen_US
dc.subjectIL-17en_US
dc.subjectrheumatoid arthritisen_US
dc.titleRheumatoid arthritis and the proinflammatory cytokine IL-17en_US
dc.typeArticleen_US
dc.identifier.doi10.3897/folmed.65.e72448-
dc.identifier.urlhttps://foliamedica.bg/article/72448/download/pdf/-
dc.identifier.urlhttps://foliamedica.bg/article/72448/download/xml/-
dc.identifier.urlhttps://foliamedica.bg/article/72448/download/pdf/-
dc.identifier.volume65-
dc.identifier.issue1-
dc.identifier.fpage53-
dc.identifier.lpage59-
item.grantfulltextopen-
item.fulltextWith Fulltext-
crisitem.author.deptFaculty of Medicine-
Appears in Collections:Faculty of Medicine: Journal Articles
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