Please use this identifier to cite or link to this item: http://hdl.handle.net/20.500.12188/29867
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dc.contributor.authorMitevski Goranen_US
dc.contributor.authorNikolovski Roberten_US
dc.contributor.authorFurnadjiski Atanasen_US
dc.contributor.authorManev, Nikolaen_US
dc.contributor.authorGeorgiev, Antonioen_US
dc.date.accessioned2024-03-28T08:55:43Z-
dc.date.available2024-03-28T08:55:43Z-
dc.date.issued2023-
dc.identifier.urihttp://hdl.handle.net/20.500.12188/29867-
dc.description.abstractMyocardial injury is when there is elevated cardiac troponin values at least one value above the upper reference limit (URL). Causes of elevated troponin can be sustained bradyarrhythmia, hypotension, shock, respiratory failure, anemia, tachyarrhythmia, hypertension, heart failure, defibrillator shocks, sepsis, chronic kidney disease, stroke, subarachnoid hemorrhage, pulmonary embolism, pulmonary hypertension, infiltrative disease (amyloidosis, sarcoidosis..), chemotherapeutic agents, critically ill patients, strenuous exercise, cardiac ablation, cardiomyopathy, coronary spasm, microvascular dysfunction, atherosclerotic plaque disruption with thrombosis, etc. When it is associated with myocardial ischemia it is designated as myocardial infarction (MI). Type 2 myocardial infarction occurs due to disturbed oxygen supply and/or unbalanced demand. There are no conditions that are characteristic for type 1 MI such as plaque, rupture and thrombosisen_US
dc.language.isoenen_US
dc.publisherMIT University Skopjeen_US
dc.relation.ispartofInternational Journal of Recent Research in Arts and Sciencesen_US
dc.subjecttype 2 MIen_US
dc.subjectcausesen_US
dc.subjectrisk factorsen_US
dc.subjecttreatmenten_US
dc.subjectprognosisen_US
dc.titleTYPE 2 MYOCARDIAL INFARCTIONen_US
dc.typeArticleen_US
item.grantfulltextopen-
item.fulltextWith Fulltext-
crisitem.author.deptFaculty of Medicine-
Appears in Collections:Faculty of Medicine: Journal Articles
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